By Alan Martin, Sam Harbison, Karen Beach, Peter Cole
A brand new version of the UK's general advent to the dangers awarded by way of ionizing radiation and the tools of safeguard and control.
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Extra info for An Introduction to Radiation Protection 6E
A considerable number of diseases are associated with recessive genes and will therefore manifest themselves only when both parents have the same recessive genes. Spontaneous mutation accounts for the fact that an appreciable fraction of the world’s population suffers from 1 of the 500 or more defects or diseases attributable to heritable effects. Radiation can induce gene mutations which are indistinguishable from naturally occurring mutations. It should be noted in passing that heat and chemicals can also cause mutations.
Genes carry the information which determines the characteristics of daughter cells. Mitosis: the process by which single cells reproduce. Meiosis: a stage in the formation of the reproductive cells – the sperm in the male and the ovum in the female. Effects of radiation on cells: inhibition of mitosis, chromosome aberrations. Acute effects: effects occurring within a few weeks of a very large exposure; owing to the depletion of cell populations. Late effects: effects occurring at later times, typically some years after exposure; main effect is cancer induction.
The range of doses from 3 to 10 Gy is often called the region of infection death. e. infection-free) environment and giving a bone marrow transfusion to stimulate white blood cell production. For doses above about 10 Gy, survival time drops abruptly to between 3 and 5 days. It remains at about this ﬁgure until much higher doses are reached. In this region, the radiation dose causes severe depletion of the cells lining the intestine. Gross damage occurs in the lining of the intestine, followed by severe bacterial invasion.
An Introduction to Radiation Protection 6E by Alan Martin, Sam Harbison, Karen Beach, Peter Cole